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mouse epidermis derived jb6 cl 41 5a cells  (ATCC)


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    ATCC mouse epidermis derived jb6 cl 41 5a cells
    Spautin‐1 significantly attenuated proliferation of melanoma cells in a time‐ and dose‐dependent manner. A, Viability analysis after the treatment of A375 and Sk‐Mel‐28 cells with different concentration of spautin‐1 (up to 20 µmol/L) for 0, 24, 48 and 72 h. (Mean values ± SEM, n = 6). B, The IC50 values of spautin‐1 in A375 and Sk‐Mel‐28 were automatically calculated by GraphPad Prism software. C, spautin‐1 inhibited A375 and Sk‐Mel‐28 cell colony formation. The number of colonies normalized to the corresponding untreated cells formed colonies. (Mean values ± SEM, n = 3) Significant differences were evaluated using a one‐way ANOVA. * P < .05 vs control group, # P < .05 vs 2.5 µmol/L. D, Non‐tumorigenic cell lines (HaCaT and <t>JB6)</t> were treated with various concentration of spautin‐1 for 72 h as indicated, and cell viability in the presence of HaCat and JB6 was measured by MTS as described in the Section and analysed by GraphPad Prism software (mean values ± SEM, n = 6)
    Mouse Epidermis Derived Jb6 Cl 41 5a Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 1018 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+epidermis+derived+jb6+cl+41+5a+cells/JB6+Cl+41-5a/pmc07171391-26-15-25
    Average 96 stars, based on 1018 article reviews
    mouse epidermis derived jb6 cl 41 5a cells - by Bioz Stars, 2026-08
    96/100 stars

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    1) Product Images from "Potent USP10/13 antagonist spautin‐1 suppresses melanoma growth via ROS‐mediated DNA damage and exhibits synergy with cisplatin"

    Article Title: Potent USP10/13 antagonist spautin‐1 suppresses melanoma growth via ROS‐mediated DNA damage and exhibits synergy with cisplatin

    Journal: Journal of Cellular and Molecular Medicine

    doi: 10.1111/jcmm.15093

    Spautin‐1 significantly attenuated proliferation of melanoma cells in a time‐ and dose‐dependent manner. A, Viability analysis after the treatment of A375 and Sk‐Mel‐28 cells with different concentration of spautin‐1 (up to 20 µmol/L) for 0, 24, 48 and 72 h. (Mean values ± SEM, n = 6). B, The IC50 values of spautin‐1 in A375 and Sk‐Mel‐28 were automatically calculated by GraphPad Prism software. C, spautin‐1 inhibited A375 and Sk‐Mel‐28 cell colony formation. The number of colonies normalized to the corresponding untreated cells formed colonies. (Mean values ± SEM, n = 3) Significant differences were evaluated using a one‐way ANOVA. * P < .05 vs control group, # P < .05 vs 2.5 µmol/L. D, Non‐tumorigenic cell lines (HaCaT and JB6) were treated with various concentration of spautin‐1 for 72 h as indicated, and cell viability in the presence of HaCat and JB6 was measured by MTS as described in the Section and analysed by GraphPad Prism software (mean values ± SEM, n = 6)
    Figure Legend Snippet: Spautin‐1 significantly attenuated proliferation of melanoma cells in a time‐ and dose‐dependent manner. A, Viability analysis after the treatment of A375 and Sk‐Mel‐28 cells with different concentration of spautin‐1 (up to 20 µmol/L) for 0, 24, 48 and 72 h. (Mean values ± SEM, n = 6). B, The IC50 values of spautin‐1 in A375 and Sk‐Mel‐28 were automatically calculated by GraphPad Prism software. C, spautin‐1 inhibited A375 and Sk‐Mel‐28 cell colony formation. The number of colonies normalized to the corresponding untreated cells formed colonies. (Mean values ± SEM, n = 3) Significant differences were evaluated using a one‐way ANOVA. * P < .05 vs control group, # P < .05 vs 2.5 µmol/L. D, Non‐tumorigenic cell lines (HaCaT and JB6) were treated with various concentration of spautin‐1 for 72 h as indicated, and cell viability in the presence of HaCat and JB6 was measured by MTS as described in the Section and analysed by GraphPad Prism software (mean values ± SEM, n = 6)

    Techniques Used: Concentration Assay, Software, Control



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    mouse epidermis derived jb6 cl 41 5a cells  (ATCC)
    96
    ATCC mouse epidermis derived jb6 cl 41 5a cells
    Spautin‐1 significantly attenuated proliferation of melanoma cells in a time‐ and dose‐dependent manner. A, Viability analysis after the treatment of A375 and Sk‐Mel‐28 cells with different concentration of spautin‐1 (up to 20 µmol/L) for 0, 24, 48 and 72 h. (Mean values ± SEM, n = 6). B, The IC50 values of spautin‐1 in A375 and Sk‐Mel‐28 were automatically calculated by GraphPad Prism software. C, spautin‐1 inhibited A375 and Sk‐Mel‐28 cell colony formation. The number of colonies normalized to the corresponding untreated cells formed colonies. (Mean values ± SEM, n = 3) Significant differences were evaluated using a one‐way ANOVA. * P < .05 vs control group, # P < .05 vs 2.5 µmol/L. D, Non‐tumorigenic cell lines (HaCaT and <t>JB6)</t> were treated with various concentration of spautin‐1 for 72 h as indicated, and cell viability in the presence of HaCat and JB6 was measured by MTS as described in the Section and analysed by GraphPad Prism software (mean values ± SEM, n = 6)
    Mouse Epidermis Derived Jb6 Cl 41 5a Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+epidermis+derived+jb6+cl+41+5a+cells/JB6+Cl+41-5a/pmc07171391-26-15-25
    Average 96 stars, based on 1 article reviews
    mouse epidermis derived jb6 cl 41 5a cells - by Bioz Stars, 2026-08
    96/100 stars
    		  Buy from Supplier

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    Spautin‐1 significantly attenuated proliferation of melanoma cells in a time‐ and dose‐dependent manner. A, Viability analysis after the treatment of A375 and Sk‐Mel‐28 cells with different concentration of spautin‐1 (up to 20 µmol/L) for 0, 24, 48 and 72 h. (Mean values ± SEM, n = 6). B, The IC50 values of spautin‐1 in A375 and Sk‐Mel‐28 were automatically calculated by GraphPad Prism software. C, spautin‐1 inhibited A375 and Sk‐Mel‐28 cell colony formation. The number of colonies normalized to the corresponding untreated cells formed colonies. (Mean values ± SEM, n = 3) Significant differences were evaluated using a one‐way ANOVA. * P < .05 vs control group, # P < .05 vs 2.5 µmol/L. D, Non‐tumorigenic cell lines (HaCaT and JB6) were treated with various concentration of spautin‐1 for 72 h as indicated, and cell viability in the presence of HaCat and JB6 was measured by MTS as described in the Section and analysed by GraphPad Prism software (mean values ± SEM, n = 6)

    Journal: Journal of Cellular and Molecular Medicine

    Article Title: Potent USP10/13 antagonist spautin‐1 suppresses melanoma growth via ROS‐mediated DNA damage and exhibits synergy with cisplatin

    doi: 10.1111/jcmm.15093

    Figure Lengend Snippet: Spautin‐1 significantly attenuated proliferation of melanoma cells in a time‐ and dose‐dependent manner. A, Viability analysis after the treatment of A375 and Sk‐Mel‐28 cells with different concentration of spautin‐1 (up to 20 µmol/L) for 0, 24, 48 and 72 h. (Mean values ± SEM, n = 6). B, The IC50 values of spautin‐1 in A375 and Sk‐Mel‐28 were automatically calculated by GraphPad Prism software. C, spautin‐1 inhibited A375 and Sk‐Mel‐28 cell colony formation. The number of colonies normalized to the corresponding untreated cells formed colonies. (Mean values ± SEM, n = 3) Significant differences were evaluated using a one‐way ANOVA. * P < .05 vs control group, # P < .05 vs 2.5 µmol/L. D, Non‐tumorigenic cell lines (HaCaT and JB6) were treated with various concentration of spautin‐1 for 72 h as indicated, and cell viability in the presence of HaCat and JB6 was measured by MTS as described in the Section and analysed by GraphPad Prism software (mean values ± SEM, n = 6)

    Article Snippet: The human malignant melanoma cell lines (A375 and SK‐Mel‐28), the human keratinocyte HaCaT and the mouse epidermis‐derived JB6 Cl 41‐5a cells were purchased from the American Type Culture Collection (ATCC).

    Techniques: Concentration Assay, Software, Control